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*PRACTICE OVER* Training camp tweets 8/6/11 **Fan Appreciation Day** **Henson Released** **Kerrigan changes jersey number to 91** P Sav Rocca reports to Redskin Park


c4man5282

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Trivia: Which college has the most players on the Redskins roster? Answer by 5pm for a chance to win 2 tickets to @Redskins vs Steelers game

dshif Daniel Shiferaw

Having a tough time getting into Redskins Park due to traffic. Wild guess: I think there'll be a good turnout today

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Performances by the #Redskins Cheerleaders and the Redskins Marching Band are coming up.

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Redskins players are due to start warming up and stretching at about 11:20 a.m. Practice starts at 12 noon.

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And a team-wide autograph session is expected to begin at 1:30 p.m. Runs from 20 minutes.

---------- Post added August-6th-2011 at 10:17 AM ----------

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Seems like a daily occurrence here at #Redskins training camp -- a jersey number change: Ryan Kerrigan is now No. 91 (previously No. 53).

Senior Bowl number

Ryan-Kerrigan-Senior-Bowl.jpg

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For those of you that don't know, micro tears in the tendons are a direct side effect of chronic anabolic steroid use. People and animals that take or are given anabolic steroids have major tendon issues down the road. Typical is the snapping of a tendon as it doesn't grow when muscles are over enlarged and the resulting tendons are stretched until they give way. LL's calves must be enormous and were most likely on the verge of snapping. In the wake of experiencing this type of injury you basically have to quit using it for a long time. You can't stretch it anymore, nor can you continue to build the attaching muscle groups. He may quit working out his legs and that will result in LL loosing a step or two. He probably would have gone to HGH only after the diagnosis as that would have helped in repairing the tendon. He doesn't look like he's lost any size from what can be seen, however the speed and burst will be gone. FYI.

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The landry recovery news is encouraging, but if he hasn't been able to run since last december, what kind of conditioning will he be in? Doesn't say god things about his speed or stamina. Guess we can't expect the return of the same landry from the beginning of last year. Thats too bad.

He will be slower and won't have the same burst. Definitely. See my other post.

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For those of you that don't know, micro tears in the tendons are a direct side effect of chronic anabolic steroid use. People and animals that take or are given anabolic steroids have major tendon issues down the road. Typical is the snapping of a tendon as it doesn't grow when muscles are over enlarged and the resulting tendons are stretched until they give way. LL's calves must be enormous and were most likely on the verge of snapping. In the wake of experiencing this type of injury you basically have to quit using it for a long time. You can't stretch it anymore, nor can you continue to build the attaching muscle groups. He may quit working out his legs and that will result in LL loosing a step or two. He probably would have gone to HGH only after the diagnosis as that would have helped in repairing the tendon. He doesn't look like he's lost any size from what can be seen, however the speed and burst will be gone. FYI.

While true microtears are also created due to normal use. And if he was overusing his body continually playing while injured (as most football players do..) then there you go.

Since the NFL is going to up their standards with HGH testing now too I won't point any fingers until people test positive.

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Rich_Tandler Rich Tandler

Looks like Henson was ready to practice but the Skins did not want him. If he hurt himself again, team liable for 2011 salary. They cut bait

---------- Post added August-6th-2011 at 10:31 AM ----------

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Redskins Marching Band arriving on the field. Of course, they're playing "Hail to the Redskins."

---------- Post added August-6th-2011 at 10:34 AM ----------

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Following the Marching Band, it's the #Redskins Cheerleaders decked out in gold outfits with burgundy trims.

washington-cheerleaders.jpg

Redskins_Cheerleaders_21.jpg

Guessing this is what they look like.

09000d5d81238dfc_gallery_600.jpg

Or do you guys like this pic better :silly:

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For those of you that don't know, micro tears in the tendons are a direct side effect of chronic anabolic steroid use. People and animals that take or are given anabolic steroids have major tendon issues down the road. Typical is the snapping of a tendon as it doesn't grow when muscles are over enlarged and the resulting tendons are stretched until they give way. LL's calves must be enormous and were most likely on the verge of snapping. In the wake of experiencing this type of injury you basically have to quit using it for a long time. You can't stretch it anymore, nor can you continue to build the attaching muscle groups. He may quit working out his legs and that will result in LL loosing a step or two. He probably would have gone to HGH only after the diagnosis as that would have helped in repairing the tendon. He doesn't look like he's lost any size from what can be seen, however the speed and burst will be gone. FYI.

From Webmd:

What is the Achilles tendon?

The Achilles tendon connects the calf muscle to the heel bone. It lets you rise up on your toes and push off when you walk or run.

What are common Achilles tendon problems?

The two main problems found in the Achilles tendon are:

Achilles tendinopathy. Achilles tendinopathy includes one of two conditions:

Tendinitis. This actually means "inflammation of the tendon," but inflammation is rarely the cause of tendon pain.

Tendinosis. This refers to tiny tears (microtears) in the tissue in and around the tendon caused by overuse. In most cases Achilles tendon pain is the result of tendinosis, not tendinitis. Some experts now use the term tendinopathy to include both inflammation and microtears. But many doctors may still use the term tendinitis to describe a tendon injury.

Achilles tendon tear or rupture. An Achilles tendon also can partially tear or completely tear (rupture) . A partial tear may cause mild or no symptoms. But a complete rupture causes pain and sudden loss of strength and movement.

Problems with the Achilles tendon may seem to happen suddenly. But usually they are the result of many tiny tears to the tendon that have happened over time.

What causes Achilles tendon problems?

Achilles tendon problems are most often caused by overuse or repeated movements. These movements can happen during sports, work, or other activities. For example, if you do a lot of pushing off or stop-and-go motions when you play sports, you can get microtears in the tendon. Microtears can also happen with a change in how long, hard, or often you exercise. Microtears in the tendon may not be able to heal quickly or completely.

Being out of shape or not warming up before exercising may also cause Achilles tendon problems. So can shoes with poor arch supports or rigid heels.

An Achilles rupture is most often caused by a sudden, forceful motion that stresses the calf muscle. This can happen during an intense athletic activity or even during simple running or jumping. Middle-aged adults are especially likely to get this kind of injury.

A rupture most often occurs in sports such as basketball, racquet sports (including tennis), soccer, and softball. A tendon already weakened by overstretching, inflammation, or small tears is more likely to rupture.

http://www.webmd.com/a-to-z-guides/achilles-tendon-problems-topic-overview

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While true microtears are also created due to normal use. And if he was overusing his body continually playing while injured (as most football players do..) then there you go.

Since the NFL is going to up their standards with HGH testing now too I won't point any fingers until people test positive.

You obviously haven't seen LL. He's not natural. That didn't happen from protein shakes!

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From Webmd:

What is the Achilles tendon?

The Achilles tendon connects the calf muscle to the heel bone. It lets you rise up on your toes and push off when you walk or run.

What are common Achilles tendon problems?

The two main problems found in the Achilles tendon are:

Achilles tendinopathy. Achilles tendinopathy includes one of two conditions:

Tendinitis. This actually means "inflammation of the tendon," but inflammation is rarely the cause of tendon pain.

Tendinosis. This refers to tiny tears (microtears) in the tissue in and around the tendon caused by overuse. In most cases Achilles tendon pain is the result of tendinosis, not tendinitis. Some experts now use the term tendinopathy to include both inflammation and microtears. But many doctors may still use the term tendinitis to describe a tendon injury.

Achilles tendon tear or rupture. An Achilles tendon also can partially tear or completely tear (rupture) . A partial tear may cause mild or no symptoms. But a complete rupture causes pain and sudden loss of strength and movement.

Problems with the Achilles tendon may seem to happen suddenly. But usually they are the result of many tiny tears to the tendon that have happened over time.

What causes Achilles tendon problems?

Achilles tendon problems are most often caused by overuse or repeated movements. These movements can happen during sports, work, or other activities. For example, if you do a lot of pushing off or stop-and-go motions when you play sports, you can get microtears in the tendon. Microtears can also happen with a change in how long, hard, or often you exercise. Microtears in the tendon may not be able to heal quickly or completely.

Being out of shape or not warming up before exercising may also cause Achilles tendon problems. So can shoes with poor arch supports or rigid heels.

An Achilles rupture is most often caused by a sudden, forceful motion that stresses the calf muscle. This can happen during an intense athletic activity or even during simple running or jumping. Middle-aged adults are especially likely to get this kind of injury.

A rupture most often occurs in sports such as basketball, racquet sports (including tennis), soccer, and softball. A tendon already weakened by overstretching, inflammation, or small tears is more likely to rupture.

http://www.webmd.com/a-to-z-guides/achilles-tendon-problems-topic-overview

Really? WebMD. OMG!

Get your head out of the sand...

Here is a real article from Steroid Encyclopedia:

Tendons

While injecting test increases protein synthesis by roughly 50 times, depending on dose and time, most bodybuilders forget that it will reduce collagen synthesis by more than 50% -- more like 80%, giving you the collagen synthesis rate of a senior citizen. Since collagen makes up tendons, bros are very prone to injury if they continue to lift very heavy, unless they cycle off T and let their collagen synthesis get back to normal. It's like having the skeletal muscle of a gorilla with the tendons of a very old man.

Winstrol increases collagen synthesis. It will give you bigger tendons. However, your body compensates for this by making them more brittle, weaker, and more prone to injury. I can't tell you how many bros work out anaerobically and become injured while on winstrol. Guys who lift in the 1-5 rep range while on winstrol, to baseball players who sprint all out from a stationary position -- winstrol should be the LAST drug they choose. Most of them like winstrol because they don't get the weight gain from it but it is very detrimental to bros who train for any sport anaerobically. Tendons tear easily on it.

Also, the drugs I mention increase collagen synthesis while also increasing collagen cross-linking integrity, making for a much stronger tendon.

Winstrol, on the other hand, will dramatically increase collagen syn, but ironically it decreases collagen cross-linking integrity, thus making a much weaker tendon.

You can plan a cycle of AAS which will increase collagen synthesis and skeletal muscle growth at the same time. The key is the drug(s) you choose.

Deca, Equipoise, Anavar, and Primobolan will ALL increase skeletal muscle while at the same time dramatically increase collagen syn and bone mass and density, leaving you with a substantially reduced chance of becoming injured than if you choose to use AAS like sus, cyp, or enth.

While testosterone will increase bone mass and density, even at supra-physiological levels, the result is weaker tendons due to inhibition of collagen syn.

To plan a cycle where the goal is to increase skeletal muscle mass/strength while at the same time increase joint/tendon/ligament strength, enough to keep up with the dramatic increase in skeletal muscle, you must choose drugs like Eq, Deca, Anavar, or Primo as the base of your cycle. Testosterone and its esters can be added to your cycle to keep levels within a 'normal' physiological range (ie, 100-200 mg/wk) but must not go above this. Since drugs like eq, deca, anavar and primo will reduce endogenous, natural levels of test, these levels may be maintained with exogenous test in the 100-200 mg/wk range. Test at this dose will not inhibit collagen syn, but paradoxically, will help increase it. It is when exogenous testosterone is used > 200 mg/wk that collagen syn is inhibited.

Deca @ 3 mg/kg a week(about 270 mg/wk for a 200 lb male) will increase procollagen III levels by 270% by week 2. Procollagen III is a primary indicator used to determine the rate of collagen syn. As you can see, deca is a very good drug at giving you everything you want -- an increase in collagen syn, an increase in skeletal muscle, and increases in bone mass and density. The one thing it does not give you is wood

Primobolan, @ 5 mg/kg, will increase collagen synthesis by roughly 180% -- less than deca and equipoise but still substantial.

Equipoise @ 3 mg/kg will increase procollagen III by approximately 340% -- slightly better than deca.

Oxandrolone has over a hundred studies documenting its effectiveness at treating patients needing rapid increases in collagen syn to enhance healing.

These drugs have longer half-lives than most other AAS, so this should be considered when timing your post cycle clomid use. Here they are:

Deca: 15 days Equipoise: 14 days Primobolan: 10.5 days

Anavar has a half-life of only 8 hours so it should not pose a problem.

GH is probably the most remarkable drug at increasing collagen synthesis. It increases collagen syn in a dose dependant manner -- the more you use, the more you will increase collagen syn. It has also demonstrated this ability in short and long term studies. From what I've read, hGH at 6 iu/day increased the collagen deposition rate by around 250% in damaged collagen structures. This result indicates that the increased biomechanical strength of wounds to collagen structures treated with biosynthetic human growth hormone was produced by an increased deposition of collagen in the collagen structures.

Eq, primo, anavar, and deca are all good -- they increase several biomakers of collagen syn -- ie, type III, II, I, procollagen markers. GH just seems to do so most dramatically.

Use of any of these drugs @ supra-physiological levels with a maintenance dose of test will increase collagen syn while at the same time increase skeletal muscle mass. Skeletal muscle mass gains will not be as dramatic as with large testosterone doses but you have to weigh the risk/reward basis for yourself. Also, these drugs do not satisfy the libido like testosterone, but that is not the point of this thread. It is only to demonstrate that you can increase skeletal muscle and collagen syn at the same time with certain AAS, the decision is up to you.

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You obviously haven't seen LL. He's not natural. That didn't happen from protein shakes!

Not quite there. Have you seen any natural bodybuilders before? For example,

http://www.realweighttraining.com/wp-content/uploads/2009/08/laynenorton.jpg

http://www.natural-muscle-mass.com/images/mike-ashley.jpg

Landry's physique isn't that insanely muscled that I think he is using. He might be using, but until there's proof I wouldn't say he's using. Most of the guys at elite level of play already have gifted genetics for muscle and recovery already, so just because they have a freaky frame doesn't mean they are using steroids.

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Rich_Campbell Rich Campbell

Fan Appreciation Day traffic entering Redskins Park is backed up half a mile south on Loudoun Co. Pkwy. If you're coming, leave now.

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Training Camp Practice Report: Aug. 6: Redskins.com's training camp practice reports come to you with news and... http://redsk.in/pSLDkC

---------- Post added August-6th-2011 at 11:01 AM ----------

This has nothing to do with the Redskins but it is a very large number.

ClaytonESPN John Clayton

By my count, the number of torn Achilles in camps around the league has now grown to 8

---------- Post added August-6th-2011 at 11:01 AM ----------

Russellmania980 Chris Russell

New #Redskins P Sav Rocca has reported to #Redskins Park

We have a punter!!!

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